The farce of “healthcare” should be obvious to any breathing, thinking human in the developed world–but surprisingly, it’s not. We watch the Cialis commercials and wonder with bemusement how the hell they got the bathtubs out there on the mountaintop–and where the hot water came from. And if that water’s cold, then don’t even bother with the manhancement drug, buddy.
We think things like this, instead of wondering why the regulatory agencies allow any drug on the market in the first place, considering the dire warnings of adverse effects–particularly the one about “don’t take this medication if you are at increased risk of death.”
Take Humira, for example. In the commercials, attractive (but not too attractive), usually middle-aged women are shown to be experiencing life again, since (insert syndrome here) started kicking their khaki-clad butt. God bless the pharmaceutical company that gave them their life back!
But wait! Not all is sunshine and puppy snuggling when it comes to treating your chronic fatiguing sorta illness that the doctors say is “autoimmune” because that’s what the rep told them over surf & turf.
What should I tell my doctor BEFORE starting HUMIRA?
Tell your doctor about all of your health conditions, including if you:
- Have an infection, are being treated for infection, or have symptoms of an infection
- Get a lot of infections or infections that keep coming back
- Have diabetes
- Have TB or have been in close contact with someone with TB, or were born in, lived in, or traveled where there is more risk for getting TB
- Live or have lived in an area (such as the Ohio and Mississippi River valleys) where there is an increased risk for getting certain kinds of fungal infections, such as histoplasmosis, coccidioidomycosis, or blastomycosis
- Have or have had hepatitis B
- Are scheduled for major surgery
- Have or have had cancer
- Have numbness or tingling or a nervous system disease such as multiple sclerosis or Guillain-Barré syndrome
- Have or had heart failure
- Have recently received or are scheduled to receive a vaccine. HUMIRA patients may receive vaccines, except for live vaccines
- Are allergic to rubber, latex, or any HUMIRA ingredients
- Are pregnant, planning to become pregnant, breastfeeding, or planning to breastfeed
- Have a baby and you were using HUMIRA during your pregnancy. Tell your baby’s doctor before your baby receives any vaccines
https://www.humira.com/important-safety-information
First I wonder, how are they allowed to give this garbage to ANYONE?
But more importantly, I wonder why nobody seems to realize that almost every one of those warnings has something to do with the bad effects of Humira, an immune-suppressing drug, on an already suppressed immune system.
Infections? Immunosuppressive.
Influenza A virus infection predisposes hosts to secondary infection with different Streptococcus pneumoniae serotypes with similar outcome but serotype-specific manifestation. Sharma-Chawla N, et al. Infect Immun. 2016.
https://www.ncbi.nlm.nih.gov/m/pubmed/27647871/
This is why doctors used to prescribe prophylactic antibiotics for kids with the flu. Now they want you to get flu vaccines and pneumonia vaccines instead. So, when you get those vaccines, cross your fingers and say a little prayer that they’re not contaminated with some type of fungus or mycotoxin. Why? Because there’s a pretty good chance that they are contaminated.
“Experimental studies conducted in various countries have shown the mycoplasma infection rate among cultures in different laboratories to vary from 15% to 80% and, in some, to even reach 100% [3, 5].”
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4207559/
…And because…TB? Fungal infections? Immunosuppressive.
“Thus, prolonged TLR2 signalling induced by M. tuberculosis lipoproteins (and, potentially, by other TLR2 agonists expressed by M. tuberculosis18) results in inhibition of MHC class II molecule expression and antigen presentation by M. tuberculosis-infected macrophages.”
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3037727/
The 19-kD antigen and protective immunity in a murine model of tuberculosis.
“The 19-kD antigen is a cell wall-associated lipoprotein present in Mycobacterium tuberculosis and in bacille Calmette-Guérin (BCG) vaccine strains. Expression of the 19-kD antigen as a recombinant protein in two saprophytic mycobacteria-M. vaccae and M. smegmatis-resulted in abrogation of their ability to confer protection against M. tuberculosis in a murine challenge model, and in their ability to prime a DTH response to cross-reactive mycobacterial antigens. Induction of an immune response to the 19-kD antigen by an alternative approach of DNA vaccination had no effect on subsequent M. tuberculosis challenge. These results are consistent with a model in which the presence of the 19-kD protein has a detrimental effect on the efficacy of vaccination with live mycobacteria. Targeted inactivation of genes encoding selected antigens represents a potential route towards development of improved vaccine candidates.”
T cells from patients with Candida sepsis display a suppressive immunophenotype.
“CONCLUSIONS: Circulating immune effector cells from patients with Candidemia display an immunophenotype consistent with immunosuppression as evidenced by T cell exhaustion and concomitant downregulation of positive co-stimulatory molecules. These findings may help explain why patients with fungal sepsis have a high mortality despite appropriate antifungal therapy. Development of immunoadjuvants that reverse T cell exhaustion and boost host immunity may offer one way to improve outcome in this highly lethal disorder.”
Even Anthony Fauci, director of the NIAID, says so, when it comes to potentially making some money on it. Like here, in his patent for IL-2 as an immune booster for immune suppressing infections like fungi and stuff.
“BACKGROUND OF THE INVENTION
“….Illustrative of specific disease states in treatment of which the present invention can be applied are HIV infection and other diseases characterized by a decrease of T-cell immunity, for example, mycobacterial infections like tuberculosis and fungal infections such as cryptococcal disease. This method also can be used in the treatment of secondary infections that occur in patients with suppressed immune systems, such as the opportunistic infections that occur in AIDS patients. …”
HepB? Reactivates with immunosuppression. https://www.ncbi.nlm.nih.gov/m/pubmed/27291888/
Major surgery? Heart failure? Better off dead?
Safety Communications > Nontuberculous Mycobacterium Infections Associated with Heater-Cooler Devices: FDA Safety Communication – http://www.fda.gov
“Through the FDA’s analysis of adverse event reports, the medical literature, and information from national and international public health agencies, we are aware that the use of heater-cooler devices has been associated with Nontuberculous ***Mycobacteria*** (NTM) infections, primarily in patients undergoing cardiothoracic surgical procedures. NTM organisms are widespread in nature and can be found in soil and water, including tap water sources. They are typically not harmful, but in rare cases may cause infections in very ill patients and/or in individuals with compromised immune systems.”
http://www.fda.gov/MedicalDevices/Safety/AlertsandNotices/ucm466963.htm
“A research study “Prolonged Outbreak of Mycobacterium chimaera Infection After Open-Chest Heart Surgery” published in March 2015 in Clinical Infectious Diseases on an outbreak in Switzerland determined that the water used in the heater-coolers became contaminated by M. chimaera based on how the fan on the heater-cooler unit aerosolized the organism into the air. While the water never came in contact with the patient, the aerosol spread by the fan directly contaminated the surgical wound. The study also found that when the heater-cooler devices were in the operating room but not turned on during the surgery, the organism was not aerosolized. Of the reported cases in this study, most have manifested as prosthetic valve endocarditis or vascular graft infections. Study authors pointed out the possibility that there may be involvement of the bone marrow causing cytopenias as well as splenomegaly. The patients in the study – ***who were not immunocompromised*** – presented with nonlocalizing symptoms, such as fever, myalgias, arthralgias, fatigue and weight loss. The mortality rate was approximately 50%.”
Lovely.
Cancer’s on the list? Well, of course. It’s an immunosuppression disease.
Then we get to the part about vaccines.
“Have recently received or are scheduled to receive a vaccine? HUMIRA patients may receive vaccines, except for live vaccines.”
Oh, really? Is that because you could actually get the virus instead of the protection if you happen to be immunosuppressed?
Paul Gisbert Auwaerter, who says you need psych intervention for your Lyme-OspA-fungal-type-antigen-immunosuppression, reported in 1999 that measles vaccine virus returns to full virulence in the gut of an immune deficient child–and it killed this particular child.
“To determine whether in vivo passage of vaccine strains of MV alters their virulence and to begin to elucidate the determinants of MV growth and virulence in vivo, we have studied the growth of vaccine-derived virus strains pMor-1 and Hu2 in thy/liv implants. These viruses had lost the attenuated Moraten phenotype and grew with kinetics similar to Ed-wt, the minimally passaged parent of the Moraten and Schwarz vaccine strains. thy/liv implant infection with pMor-1 and Hu2 resulted in high levels of virus production, suggesting that prolonged growth of live attenuated MV vaccine in human tissue selects for a virus adapted to grow in human tissues in vivo. Our data suggest that the adverse outcomes associated with immunization of patients suffering from congenital and acquired immunodeficiency syndromes are due to the emergence of an MV strain with increased virulence in a host unable to mount a sufficient immune response to clear the originally inoculated vaccine virus. This situation is mimicked in the SCID-hu mouse. Sequence analyses of pMor-1 H and M and other isolates derived from immunodeficient patients demonstrate that these human tissue-passaged vaccine isolates are highly related to parent vaccine strains (1, 15).”
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC112900/#__ffn_sectitle
Inject some live viruses when some live viruses are already there?
“This study shows that simultaneous coinfection can cause unpredictable and highly variable alterations in T cell responses that are not identified in single-virus studies and that these alterations can result in loss of immune protection and enhanced disease upon reexposure to that pathogen.”
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4621125/
Yeah, enhanced disease. Nice.
The warning I find most intriguing, however, is the last one:
“Have a baby and you were using HUMIRA during your pregnancy. Tell your baby’s doctor before your baby receives any vaccines”
Let’s take a look at why this is recommended. First of all, understand that unfortunately, telling your baby’s doctor won’t do a bit of good. Why?
- Because your baby’s doctor is getting kickbacks for administering vaccines, and
- Because your baby’s doctor doesn’t have a clue about any of the previous 1200 or so words.
If you used Humira during your pregnancy, and if it were possible to have a pediatrician who understood that Humira is immunosuppressive, it would be important for said pediatrician to know that you were immunosuppressed. But said pediatrician would also have to understand that herpesviruses (EBV, CMV, etc.), which are nearly ubiquitous among adults, are reactivated in an immunosuppressed host. I shouldn’t have to cite this, because SHINGLES, but here you go: https://www.ncbi.nlm.nih.gov/m/pubmed/?term=herpes+immunosuppression
Additionally, such pediatrician would have to understand that these herpesviruses can be transmitted to the fetus. Some do know this last bit. It’s well known that congenital CMV can have some nasty outcomes. Go ahead and search PubMed. https://www.ncbi.nlm.nih.gov/m/pubmed/?term=congenital+herpes
In fact, look up TORCH syndrome. It’s a thing. https://en.m.wikipedia.org/wiki/TORCH_syndrome
So, if you’re on Humira while preggo it would be a shame for your baby to come out with a bunch of infections and then add some live virus vaccines to the mix. Synergy. It’s another thing.
I mean, really. Isn’t it obvious at this point that any number of factors could negatively impact a child’s immune status, thereby making him susceptible to the live vaccine viruses? Isn’t it obvious by now that a contaminated live virus vaccine can be that one-two punch of immune-suppressing contaminant and live, neurotropic virus? High-pitched screaming. Fever. Encephalitis. Seizures. SIDS. Brain damage. Gut dysregulation, i.e. immunosuppression. Autism.
Yes, it’s a thing.
Live vaccine viruses crossing the blood-brain barrier might be a thing. Just saying.
TLR2-mediated leukocyte trafficking to the developing brain. Mottahedin A, et al. J Leukoc Biol. 2016.
“Inflammation is a significant risk factor for brain injury in the perinatal period. In this study, we tested the hypothesis that activation of peripheral TLR induces inflammation in the brain, including leukocyte trafficking. Postnatal day 8 mice were injected intraperitoneally with a TLR1/2 (Pam3CSK4, P3C), TLR2/6 (FSL-1) or TLR4 (LPS) agonist, and the peripheral and central cytokine and chemokine response was determined. Infiltration of immune cells to the CSF and brain was examined by flow cytometry, and brain permeability was investigated by radioactively labeled sucrose. We report that peripheral administration of P3C to neonatal mice induces significant influx of leukocytes, mainly neutrophils and monocytes, to the CSF and brain. Infiltration of leukocytes was TLR2 and MyD88 dependent, but largely absent after administration of LPS or FSL-1. PC3-mediated accumulation of immune cells in the brain was observed in classic CNS-leukocyte gateways, the subarachnoid space and choroid plexus, as well as in the median eminence. Although P3C and LPS induced a similar degree of peripheral inflammatory responses, P3C provoked a distinct brain chemokine response and increased permeability, in particular, of the blood-CSF barrier. Collectively, our results do not support the hypothesis that TLR activation, in general, induces immune cell infiltration to the brain. Instead, we have discovered a specific TLR2-mediated mechanism of CNS inflammation and leukocyte invasion into the neonatal brain. This interaction between peripheral and central immune responses is to a large extent via the blood-CSF barrier.”
https://www.ncbi.nlm.nih.gov/m/pubmed/27493242/
Oh, but wait…the makers of Humira forgot to list a few immune suppressors:
Virus reactivation: a panoramic view in human infections, Future Virol. 2011 Apr; 6(4): 451–463. Christopher M Traylen,1 Hersh R Patel,1 Wylder Fondaw,1 Sheran Mahatme,2 John F Williams,1 Lia R Walker,1 Ossie F Dyson,1 Sergio Arce,3 and Shaw M Akula1,†
“Reactivation is the mechanism whereby a latent virus that has infected a host cell switches to a lytic stage, undergoing productive viral replication and allowing the virus to spread. Viral reactivation is associated with several stress factors [1], including viral infection (with other viruses), nerve trauma, physiologic and physical changes (e.g., fever, menstruation and exposure to sunlight) and immunosuppression (as in cytomegalovirus [CMV] disease). However, increasing evidence suggest that reactivation frequently occurs in the absence of such stimuli. This premise asserts that viruses are continuously shed, but that reactivation only occurs when local immunity is compromised by stimuli such as fever, menstruation and exposure to sunlight [2]. Generally, a lytic cycle of virus infection in vitro can be induced in cells harboring a latent virus genome by treatment with 12-O-tetradecanoyl phorbol-13-acetate (TPA) [3]. Although we have an idea of the potential stimuli that may trigger virus reactivation from latency, the critical molecular factor (SWITCH) that triggers virus reactivation is still not clear.”
And, finally, let’s not forget that antigens that are shed by the Lyme parasite are immunosuppressive.
Suppression of Long-Lived Humoral Immunity Following Borrelia burgdorferi Infection, Nicole Baumgarth, et al
http://journals.plos.org/plospathogens/article?id=10.1371/journal.ppat.1004976
Clearly babies and young children are most vulnerable to this mechanism, but the main takeaway here is that IMMUNOSUPPRESSION IS A THING. It’s a very serious thing that the drug companies admit only because prescription drugs present some liability issues, whereas with vaccines there are none. They warn *you*, the “healthcare consumer,” to talk to your clueless doctor about the contraindications, and that covers them. Surely they’d like to change that, though, and keep us all as clueless as the doctors who they “educate” with free lunches and exotic vacation-conferences.
The CDC sure doesn’t want us to know that immunosuppression is a thing. If they were OK with us knowing about it, they wouldn’t waste so much taxpayer money on bogus studies to obfuscate the immunosuppression diseases of Lyme, ME/CFS, fibromyalgia and Gulf War Illness. Like the one where CDC officer Suzanne Vernon threw out the red blood cells (to which mycoplasma adhere, obstructing the transfer of oxygen–causing fatigue) when tasked with “proving” that mycoplasma weren’t the cause of fatigue. http://jmm.microbiologyresearch.org/content/journal/jmm/10.1099/jmm.0.05316-0
Or Lyme perp Mark Klempner not actually retreating patients in his $4.7mil retreatment study that is the basis of the current Lyme treatment guidelines. http://www.nejm.org/doi/full/10.1056/NEJM200107123450202
(Just to name a couple.)
In the early days of AIDS the sh!tty government created the acronym “GRID”–“Gay Related Immune Deficiency” to stigmatize the patients and their immunosuppression disease. Now, apparently, they’ve decided that ignoring the masses with our trashed immune systems is the best defense for their precious vaccines ($24 billion) and cancer ($124 billion) industries.
They don’t have a derogatory term for us, because they’d rather we simply die a long, slow, graceful Pharma-ful death. Besides, they’re pretty much killing everyone here…so “gay” or “female” or “purple” or simply “ugly” doesn’t apply. So I’ll just call this new form of immunosuppression “Greed Related Immune Deficiency.”
Yep, they’re letting Lyme, ME/CFS & fibromyalgia patients wither away, war veterans commit suicide, babies become brain damaged for life, all out of greed…and possibly Fear that the furious backlash from the entire vaccine-abused world would destroy this country once and for all. FRID just doesn’t have the same ring to it, though.
How can that be? We have the best scientists in the world! The best research institutions!
Have a look at the list of Institutes that make up the National Institutes of Health. They run the gamut from the National Heart, Lung and Blood Institute (NHLBI) to the National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS) to the National Institute of Allergy and Infectious Diseases (NIAID). There is no National Institute of Immune Deficiency Diseases. They simply won’t acknowledge any immune deficiency unless it’s AIDS.
With all the myriad ways to become either temporarily or permanently immunosuppressed, including the vaccines themselves, there’s no reason anyone should be getting jabbed with that gunk. You don’t have to believe me. Take it from the makers of Humira. Oh, and just in case you’re a breastfeeding mother who decides to experiment with Cialis, there’s a slight chance that stuff is excreted in breast milk. Just FYI. Cause you can’t make this stuff up.
Categories: Immunosuppression Diseases, Vaccines
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Thank you ❤️
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